Search results for " Atherogenic"

showing 9 items of 9 documents

NAFLD and Atherosclerosis Are Prevented by a Natural Dietary Supplement Containing Curcumin, Silymarin, Guggul, Chlorogenic Acid and Inulin in Mice F…

2017

Non-alcoholic fatty liver disease (NAFLD) confers an increased risk of cardiovascular diseases. NAFDL is associated with atherogenic dyslipidemia, inflammation and renin-angiotensin system (RAS) imbalance, which in turn lead to atherosclerotic lesions. In the present study, the impact of a natural dietary supplement (NDS) containing Curcuma longa, silymarin, guggul, chlorogenic acid and inulin on NAFLD and atherosclerosis was evaluated, and the mechanism of action was examined. C57BL/6 mice were fed an HFD for 16 weeks; half of the mice were simultaneously treated with a daily oral administration (os) of the NDS. NAFLD and atherogenic lesions in aorta and carotid artery (histological analys…

0301 basic medicineMaleAngiotensinogenAdministration OralSettore BIO/09 - Fisiologiachemistry.chemical_compoundMice0302 clinical medicineNon-alcoholic Fatty Liver DiseasePlant GumsCommiphorachemistry.chemical_classificationNutrition and Dieteticsnon-alcoholic fatty liver disease; atherogenic lesions; diet-induced obesity; natural dietary supplement; renin-angiotensin system imbalance; Profiler PCR arrayAngiotensin IIFatty liverInulinNeoplasm Proteinsrenin-angiotensin system imbalance030211 gastroenterology & hepatologyatherogenic lesionmedicine.symptomChlorogenic AcidSilymarinmedicine.medical_specialtynatural dietary supplementCurcumindiet-induced obesityProfiler PCR array; atherogenic lesions; diet-induced obesity; natural dietary supplement; non-alcoholic fatty liver disease; renin-angiotensin system imbalanceInflammationBiologyDiet High-FatFatty Acid-Binding ProteinsArticle03 medical and health sciencesInternal medicineRenin–angiotensin systemmedicineAnimalsRNA MessengerProfiler PCR arrayatherogenic lesionsPlant ExtractsFatty acidLipid metabolismmedicine.diseaseAtherosclerosisLipid MetabolismMice Inbred C57BL030104 developmental biologyEndocrinologychemistryGene Expression RegulationSuppressor of Cytokine Signaling 3 ProteinDietary SupplementsCurcuminSteatosisDyslipidemiaFood ScienceNutrients
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Overexpression of human hepatic lipase and ApoE in transgenic rabbits attenuates response to dietary cholesterol and alters lipoprotein subclass dist…

1999

Abstract —The effect of the expression of human hepatic lipase (HL) or human apoE on plasma lipoproteins in transgenic rabbits in response to dietary cholesterol was compared with the response of nontransgenic control rabbits. Supplementation of a chow diet with 0.3% cholesterol and 3.0% soybean oil for 10 weeks resulted in markedly increased levels of plasma cholesterol and VLDL and IDL in control rabbits as expected. Expression of either HL or apoE reduced plasma cholesterol response by 75% and 60%, respectively. The HL transgenic rabbits had substantial reductions in medium and small VLDL and IDL fractions but not in larger VLDL. LDL levels were also reduced, with a shift from larger, m…

Apolipoprotein EMalemedicine.medical_specialtyVery low-density lipoproteinTransgeneLipoproteinsCholesterol VLDLHypercholesterolemiaGene ExpressionPathogenesisAnimals Genetically ModifiedCholesterol Dietarychemistry.chemical_compoundApolipoproteins EInternal medicinemedicineAnimalsHumansTransgenesParticle SizeApolipoproteins BLagomorphabiologyCholesterolCholesterol HDLLipasebiology.organism_classificationEndocrinologyCholesterolchemistrylipoproteins apoE hepatic lipase rabbits transgeneLiverDiet Atherogeniclipids (amino acids peptides and proteins)Hepatic lipaseRabbitsCardiology and Cardiovascular MedicineLipoproteinArteriosclerosis, thrombosis, and vascular biology
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Immunopathogenesis of atherosclerosis: endotoxin accelerates atherosclerosis in rabbits on hypercholesterolemic diet.

2001

Background—On the basis of our concept that atherosclerosis has an immunopathological background, we tested whether activation of the innate immune system influences its progression.Methods and Results—Hypercholesterolemic (0.5% wt/wt diet) rabbits received either repeated intravenous injections of endotoxin (Escherichia colilipopolysaccharide 1.25 to 2.5 μg, once per week) or a self-limiting cutaneousStaphylococcus aureusinfection with or without a quinolone antibiotic. Measured laboratory parameters, including LDL and HDL cholesterols, were similar in the different groups of hypercholesterolemic animals. All endotoxin-treated animals developed transient episodes of fever after endotoxin a…

ArteriosclerosisInnate immunologyHypercholesterolemiaTriglycerides bloodPathogenesisCholesterol Dietarychemistry.chemical_compoundImmunityPhysiology (medical)MedicineAnimalsAortaTriglyceridesInnate immune systemCholesterolbusiness.industryDisease progressionCholesterol HDLCholesterol LDLImmunity InnateCholesterol bloodEndotoxinsDisease Models AnimalCholesterolchemistryImmunologyDisease ProgressionDiet AtherogenicFemaleStaphylococcal Skin InfectionsRabbitsCardiology and Cardiovascular MedicinebusinessCirculation
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Anti-atherogenic Effects of 17β-Estradiol

2013

Estrogens are secreted primarily by the ovaries and placenta, by the testes in men and also produced by peripheral steroidogenic conversion. The 3 major naturally occurring estrogens are: 17β-estradiol (E2), estrone and estriol, of which E2 is the predominant and most active. The actions of E2 are mediated by at least 3 different receptors - the classical ERs (ERα and ERβ) and G-protein coupled receptor 30 (GPR30). E2 signaling in cardiomyocytes involves ERα- and ERβ-independent pathways, and treatment with the E2 receptor antagonists (Selective Estrogen Receptor Modulators- SERMs), which are agonists of GPR30, inhibits cardiac cell growth. Effects of E2 in preventing endothelial dysfunctio…

MaleSelective Estrogen Receptor Modulatorsmedicine.medical_specialtyVascular smooth muscleEndotheliummedicine.drug_classEndocrinology Diabetes and MetabolismOvariectomyClinical BiochemistryInflammation030204 cardiovascular system & hematologyBiologyBiochemistry03 medical and health sciencesestrogen 17β-estradiol atherogenic factors atherosclerosis0302 clinical medicineEndocrinologyRisk FactorsInternal medicinemedicineAnimalsHumansEndothelial dysfunctionReceptorEstradiolBiochemistry (medical)Estrogen Replacement TherapyGeneral Medicinemedicine.diseaseAtherosclerosis3. Good healthmedicine.anatomical_structureEndocrinologySelective estrogen receptor modulatorEstrogen030220 oncology & carcinogenesisDiet AtherogenicFemalemedicine.symptomGPER
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The predictive role of atherogenic dyslipidemia in subjects with non-coronary atherosclerosis

2009

Abstract Background Recent findings have suggested that subjects with non-coronary atherosclerosis may show elevated prevalence of atherogenic dyslipidemia, including higher triglyceride levels, reduced HDL-cholesterol concentrations and increased levels of small, dense low-density lipoproteins (LDL). These three lipid abnormalities constitute the so-called “atherogenic-lipoprotein-phenotype” (ALP) but its predictive role in these patients still remains to be established. Methods We performed a 2-year follow-up study to assess clinical and biochemical predictors of cardiovascular events in 44 male patients (64 ± 5 years, BMI: 27 ± 3), 26 with peripheral arterial disease and 18 with abdomina…

Malemedicine.medical_specialtyMultivariate analysis1303 BiochemistryClinical Biochemistry10265 Clinic for Endocrinology and Diabetology610 Medicine & health1308 Clinical Biochemistry2704 Biochemistry (medical)Biochemistrychemistry.chemical_compoundRisk Factorsatherogenic dyslipidemiaInternal medicineDiabetes mellitusmedicineHumansFamily historyCoronary atherosclerosisDyslipidemiasPeripheral Vascular DiseasesUnivariate analysisTriglyceridebusiness.industryBiochemistry (medical)Cholesterol LDLGeneral MedicineMiddle AgedAtherosclerosismedicine.diseaseAbdominal aortic aneurysmPeripheralPhenotypechemistryCase-Control StudiesMultivariate AnalysisCardiologyAtherosclerosis HDL-cholesterol Triglycerides Small dense LDL Atherogenic lipoprotein phenotypebusinessAortic Aneurysm AbdominalFollow-Up Studies
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Cilostazol and atherogenic dyslipidemia: a clinically relevant effect

2011

Cilostazol is a reversible, selective inhibitor of PDE3A able to significantly improve walking distance in patients with intermittent claudication. However, beyond its antiplatelet and vasodilator properties, cilostazol seems to have significant effects on atherogenic dyslipidemia.The effects of cilostazol on plasma lipids, lipoproteins, apolipoproteins and postprandial lipemia are reviewed. A literature search (using Medline and Scopus) was performed up to 24 October 2010. The authors also manually reviewed the references of selected articles for any pertinent material.Cilostazol is able to significantly lower plasma triglyceride levels, with a concomitant increase in high-density lipoprot…

medicine.medical_specialtyApolipoprotein BTetrazolescilostazol atherogenic dyslipidemiaPhosphodiesterase 3 InhibitorsPeripheral Arterial Diseasechemistry.chemical_compoundDiabetes mellitusInternal medicineHumansMedicinePharmacology (medical)DyslipidemiasPharmacologymedicine.diagnostic_testbiologybusiness.industryCholesterolGeneral MedicineAtherosclerosismedicine.diseaseLipidsCyclic Nucleotide Phosphodiesterases Type 3Intermittent claudicationCilostazolCilostazolPostprandialEndocrinologyDiabetes Mellitus Type 2chemistrybiology.proteinlipids (amino acids peptides and proteins)medicine.symptombusinessLipid profileLipoproteinmedicine.drug
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Complement C6 deficiency protects against diet-induced atherosclerosis in rabbits.

1998

Abstract —Low-density lipoprotein (LDL) can be transformed to an atherogenic moiety by nonoxidative, enzymatic degradation. Enzymatically degraded LDL induces macrophage foam cell formation, provokes release of cytokines, and also activates complement. To determine whether complement activation may contribute to atherogenesis, 6 pairs of homozygous C6-deficient rabbits and their non–C6-deficient heterozygous siblings were fed a cholesterol-rich diet for 14 weeks. Cholesterol levels and plasma lipoprotein profiles of the animals in the C6-competent and C6-deficient groups did not significantly differ, and the high density lipoprotein and LDL cholesterol ratios at the end of the experiment w…

medicine.medical_specialtyHeterozygoteArteriosclerosisBiologyPathogenesisCholesterol Dietarychemistry.chemical_compoundHigh-density lipoproteinInternal medicinemedicine.arterymedicineMacrophageAnimalsComplement ActivationFoam cellAortaCholesterolHomozygoteComplement systemComplement C6EndocrinologychemistryImmunologyDiet AtherogenicRabbitsCardiology and Cardiovascular MedicineLipoproteinArteriosclerosis, thrombosis, and vascular biology
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Therapy with non-invasive ventilation in patients with obstructive sleep apnoea: effects on atherogenic lipoprotein phenotype.

2009

Patients with obstructive sleep apnoea are at increased risk of atherosclerotic morbidity and mortality. Abnormalities in lipid metabolism that occur in response to chronic intermittent hypoxia in patients with sleep-disordered breathing may increase the cardiovascular risk in an already susceptible population. Atherogenic lipoprotein phenotype and small, dense LDL have an independent predictive role for future cardio- and cerebro-vascular events in patients with the metabolic syndrome. Therefore, testing the hypothesis that therapy of obstructive sleep apnoea may reduce atherogenic lipoprotein phenotype might have significant clinical implications. We suggest that abolition of obstructive …

medicine.medical_specialtyLipoproteinsmedicine.medical_treatmentmedicine.disease_causeInternal medicinemedicineHumansContinuous positive airway pressureOxygen saturation (medicine)Sleep Apnea Obstructivebusiness.industryobstructive sleep apnoea Atherogenic lipoproteinthe metabolic syndrome cardiovascular riskLipid metabolismGeneral MedicineAtherosclerosismedicine.diseaseRespiration ArtificialSleep in non-human animalsEndocrinologyCirculatory systemBreathingCardiologyMetabolic syndromebusinessaterosclerosiOxidative stress
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Effects “beyond-cholesterol” of statins: simvastatin reduces the levels of atherogenic lipoproteins in patients with premature coronary artery disease

2002

statins simvastatin atherogenic lipoproteins coronary artery disease
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